Trump Admin to Officially Revoke California’s Waiver to Set Stricter Emissions Standards
Revoking California's waiver is part of a broader effort to roll back Obama-era fuel efficiency standards for cars and light trucks. In a draft proposal released in August of 2018, the administration had promised to replace the Obama-era requirement that cars average 54.5 miles per gallon by 2025 with a much less ambitious target of around 37 miles per gallon. It also said it would revoke California's ability to set its own standards, so that automakers would not have to build for two national markets.
But the reduced standards, which would allow nearly six billion tons of carbon dioxide pollution to enter the atmosphere, have been delayed as staff work to justify them scientifically, legally and technically, The New York Times explained. So the administration decided to move forward with revoking California's waiver.
"Trump has married his administration-wide hostility to the environment to his personal vendetta against California," Safe Climate Campaign Director Dan Becker told The New York Times.
President Trump’s attack on California and other states threatens to make America polluted again. It rejects cleaner, less-polluting vehicles in favor of pollution-spewing Trump-mobiles for urban cowboys hauling lattes home from Starbucks. #cleanercarshttps://t.co/cZtxFzXQ7l pic.twitter.com/Aj9D0gZvsQ— Safe Climate Campaign (@safe_climate) September 17, 2019
The move is likely to set off a legal battle that could go all the way to the Supreme Court. Part of the reason that the administration moved forward with revoking the waiver is so that it would have a chance to defend the decision in the nation's highest court before a potential election loss in 2020. The New York Times explained the stakes of that potential fight:
Legal experts said that if Mr. Trump's move was ultimately held up by the Supreme Court, it could permanently block states from regulating vehicle greenhouse gas pollution. If it was rejected by the Supreme Court, it would allow states to set separate tailpipe pollution standards from those set by the federal government.
The outcome could split the United States auto market, with some states adhering to stricter pollution standards than others. For automakers, that would be a nightmare.
Thirteen states, representing one third of the U.S. auto market, currently follow California's standards.
For its part, California intends to fight.
"While the White House has abdicated its responsibility to the rest of the world on cutting emissions and fighting global warming, California has stepped up," Gov. Gavin Newsom told The Washington Post. "It's a move that could have devastating consequences for our kids' health and the air we breathe, if California were to roll over. But we will not."
Hi, @realDonaldTrump. Let me break it down in simple terms for you...— Gavin Newsom (@GavinNewsom) September 17, 2019
-Protects our air & health
-Is good for the economy
-Allows families to pay less at the pump
We’re here in the 21st century. Let us know when you decide to join us. https://t.co/gZzUjXeFxo
The administration was scheduled to announce the new policy Wednesday, but delayed the announcement by at least one day after the news broke Tuesday, The Washington Post reported.
"In the very near future, the Trump administration will begin taking the steps necessary to establish one set of national fuel-economy standards," Wheeler said, as POLITICO reported. "We embrace federalism and the role of the states, but federalism does not mean that one state can dictate standards for the nation," Wheeler continued.
The revoked waiver was also partly retaliation for a deal California struck with four automakers this summer. Ford, Honda, Volkswagen and BMW of North America agreed to emissions standards of 51 miles per gallon by 2026. The EPA and Transport Department criticized the move, and the Justice Department launched an anti-trust investigation into the deal, POLITICO reported.
"It's clear that the president felt very strongly about this," Thomas J. Pyle, president of pro-fossil fuel think tank the Institute for Energy Research, told The New York Times. "The actions of California and the other auto companies probably firmed up or hardened the president's resolve on this issue, so here we go."
But environmental experts pointed out that the battle between California and the Trump administration could have global consequences.
Obama-era EPA air official Janet McCabe told POLITICO that climate change "is a 'y'all come' situation, where we need all the reasonable programs that we can get in place. And this is one of the most reasonable and impactful across the board," she said.
EcoWatch Daily Newsletter
Monday is World Oceans Day, but how can you celebrate our blue planet while social distancing?
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By Jacob L. Steenwyk and Antonis Rokas
From the mythical minotaur to the mule, creatures created from merging two or more distinct organisms – hybrids – have played defining roles in human history and culture. However, not all hybrids are as fantastic as the minotaur or as dependable as the mule; in fact, some of them cause human diseases.
When Looking Through a Microscope Isn’t Close Enough.<p>For the last few years, <a href="http://www.rokaslab.org/" target="_blank">our team at Vanderbilt University</a>, <a href="https://www.researchgate.net/lab/Gustavo-Goldman-Lab" target="_blank">Gustavo Goldman's team at São Paulo University in Brazil</a> and many other collaborators around the world have been collecting samples of fungi from patients infected with different species of <em>Aspergillus</em> molds. One of the species we are particularly interested in is <a href="https://doi.org/10.1006/rwgn.2001.0082" target="_blank"><em>Aspergillus nidulans</em>, a relatively common and generally harmless fungus</a>. Clinical laboratories typically identify the species of <em>Aspergillus</em> causing the infection by examining cultures of the fungi under the microscope. The problem with this approach is that very closely related species of <em>Aspergillus</em> tend to look very similar in their broad morphology or physical appearance when viewing them through a microscope.</p><p>Interested in examining the varying abilities of different <em>A. nidulans</em> strains to cause disease, we decided to analyze their total genetic content, or genomes. What we saw came as a total surprise. We had not collected <em>A. nidulans</em> but <em>Aspergillus latus</em>, a close relative of <em>A. nidulans</em> and, as we were to soon find out, <a href="https://doi.org/10.1016/j.cub.2020.04.071" target="_blank">a hybrid species that evolved through the fusion of the genomes</a> of two other <em>Aspergillus</em> species: <em>Aspergillus spinulosporus</em> and an unknown close relative of <em>Aspergillus quadrilineatus</em>. Thus, we realized not only that these patients harbored infections from an entirely different species than we thought they were, but also that this species was the first ever <em>Aspergillus</em> hybrid known to cause human infections.</p>
Several Different Fungal Hybrids Cause Human Disease.<p>Hybrid fungi that can cause infections in humans are well known to occur in several different lineages of single-celled fungi known as yeasts. Notable examples include multiple different species of <a href="https://doi.org/10.1002/yea.3242" target="_blank">yeast hybrids</a> that cause the human diseases <a href="https://rarediseases.info.nih.gov/diseases/6218/cryptococcosis" target="_blank">cryptococcosis</a> and <a href="https://www.cdc.gov/fungal/diseases/candidiasis/index.html" target="_blank">candidiasis</a>. Although pathogenic yeast hybrids are well known, our discovery that the <em>A. latus</em> pathogen is a hybrid is a first for molds that cause disease in humans.</p>
(Left) Candida yeasts live on parts of the human body. Imbalance of microbes on the body can allow these yeasts, some of which are hybrids, to grow and cause infection. (Right) Cryptococcus yeasts, including ones that are hybrids, can cause life-threatening infections in primarily immunocompromised people. Centers for Disease Control and Prevention<p><a href="https://doi.org/10.1371/journal.ppat.1008315" target="_blank">Why certain <em>Aspergillus</em> species are so deadly</a> while others are harmless remains unknown. This may in part be because <a href="https://doi.org/10.1016/j.fbr.2007.02.007" target="_blank">combinations of traits, rather than individual traits</a>, underlie organisms' ability to cause disease. So why then are hybrids frequently associated with human disease? Hybrids inherit genetic material from both parents, which may result in new combinations of traits. This may make them more similar to one parent in some of their characteristics, reflect both parents in others or may differ from both in the rest. It is precisely this mix and match of traits that hybrids have inherited from their parental species that <a href="https://www.nytimes.com/2010/09/14/science/14creatures.html" target="_blank">facilitates their evolutionary success</a>, including their ability to cause disease.</p>
The Evolutionary Origin of an Aspergillus Hybrid.<p>Multiple evolutionary paths can lead to the emergence of hybrids. One path is through mating, just as the horse and donkey mate to create a mule. Another path is through the merging or fusion of genetic material from cells of different species.</p><p>It is this second path that appears to have been taken by our fungus. <em>A. latus</em> appears to have two of almost everything compared to its parental species: twice the genome size, twice the total number of genes and so on. But unlike other hybrids, which are often sterile like the mule, we found that <em>A. latus</em> is capable of reproducing both asexually and sexually.</p><p>But how distinct were the parents of <em>A. latus</em>? By comparing the parts contributed by each parent in the <em>A. latus</em> genome, we estimate that its parents are approximately 93% genetically similar, which is about as related as we humans are with lemurs. In other words, <em>A. latus</em>, an agent of infectious disease, is the fungal equivalent of a human-lemur hybrid.</p>
How A. Latus Differs From its Parents.<p>Elucidating the identity of closely related fungal pathogens and how they differ from each other in infection-relevant characteristics is a key step toward reducing the burden of fungal disease. For example, we found that <em>A. latus</em> was three times more resistant than <em>A. nidulans</em>, the species it was originally identified as using microscopy-based methods, to one of the most common antifungal drugs, <a href="https://www.drugbank.ca/drugs/DB00520" target="_blank">caspofungin</a>. This result provides a clear example of the potential importance of accurate identification of the <em>Aspergillus</em> pathogen causing an infection.</p><p>We also examined how <em>A. latus</em> and <em>A. nidulans</em> interact with cells from our immune system. We found that immune cells were less efficient at combating <em>A. latus</em> compared to <em>A. nidulans</em>, suggesting the hybrid fungus may be trickier for our immune systems to identify and destroy.</p><p>In the midst of the COVID-19 pandemic, our quest to understand <em>Aspergillus</em> pathogens is becoming more urgent. Growing evidence suggests that <a href="https://doi.org/10.1111/myc.13096" target="_blank">a fraction of COVID-19 patients are also infected with <em>Aspergillus</em>.</a> More worrying is that these <a href="https://doi.org/10.3201/eid2607.201603" target="_blank">secondary <em>Aspergillus</em> infections</a> can worsen the clinical outcomes for those infected with the novel coronavirus. That being said, we stress that little is known about <em>Aspergillus</em> infections in COVID-19 patients due to a lack of systematic testing, and none of the infections identified so far appear to have been caused by hybrids.</p><p>So, when it comes to hybrids, some are fantastic (the minotaur), some are helpful (the mule) and some are dangerous (<em>Aspergillus latus</em>). Understanding more about the biology of <em>Aspergillus latus</em> may help in our understanding of how microbial pathogens arise and how to best prevent and combat their infections.</p>
This Saturday, June 6, marks National Trails Day, an annual celebration of the remarkable recreational, scenic and hiking trails that crisscross parks nationwide. The event, which started in 1993, honors the National Trail System and calls for volunteers to help with trail maintenance in parks across the country.
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By John Letzing
This past Wednesday, when some previously hard-hit countries were able to register daily COVID-19 infections in the single digits, the Navajo Nation – a 71,000 square-kilometer (27,000-square-mile) expanse of the western US – reported 54 new cases of what's referred to locally as "Dikos Ntsaaígíí-19."
The Navajo Nation covers the corners of three different states. Google Maps
Growing Contribution<img lazy-loadable="true" src="https://assets.rebelmouse.io/eyJhbGciOiJIUzI1NiIsInR5cCI6IkpXVCJ9.eyJpbWFnZSI6Imh0dHBzOi8vYXNzZXRzLnJibC5tcy8yMzM3NDY5Ny9vcmlnaW4ucG5nIiwiZXhwaXJlc19hdCI6MTY0NjM4MTgyM30.IuQTKQs1stvYYKD6vaVTrqAyoBsUG0BhDvlhxsyKwPA/img.png?width=980" id="02a05" class="rm-shortcode" data-rm-shortcode-id="2841f82b1785df5d5ed7bf64d3bb882b" data-rm-shortcode-name="rebelmouse-image" />
World Economic Forum
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Scuba divers around the world are holding their metaphorical breath to see if a coronavirus infection affects the ability to dive.
DAN medical experts explained the difference between normal lungs, on the left, and "very serious lungs caused by COVID-19," on the right. Matias Nochetto / Divers Alert Network (DAN)
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