Thank You Trump for Making Climate a Breaking News Story
Donald Trump has done what Al Gore, Jim Hansen, climate scientists, the Sierra Club and the rest of the environmental movement could never do—make climate disruption breaking cable TV news. Trump's histrionic, largely symbolic and recklessly self-destructive decision to abandon the Paris climate agreement means, among other things, that far more Americans know about the Paris climate agreement this morning than 24 hours ago. Never has climate dominated a news cycle as it did Thursday—even when the Paris agreement was signed by all of the world, (Nicaragua and Syria excepted).
Trump did this by providing the climate crisis with what it lacked—a single villain. He has now cast himself, for most of the global community, as a James Bond style villain, a Dr. No or Goldfinger, plotting global destruction for personal power and gain. But he is, in reality, President of the United States—not a character in a movie. And in the real world the response to villainy doesn't come from a heroic special agent but through the collaborative response of a wide array of actors—other countries, U.S. cities, many of America's most powerful states, and hundreds of businesses and civil society organizations.
22 Awesome Responses to Trump's Announcement on Paris Agreement https://t.co/WXl6FSx18D— Josh Fox (@Josh Fox)1496354854.0
Trump's decision to abandon Paris has catalyzed, amplified and intensified this response. We actually, ironically, owe him a favor.
Let's begin with the depressing decision, and then look at the hope we can all take from the resistance.
Trump's decision to use the established process to exit Paris, but to remain in the underlying Rio Treaty on climate, means it will take four years for the U.S. to out—and the American people will have a chance to vote for the next President before we leave. So this announcement is largely symbolic. Trump also made clear that he would not honor the commitments President Obama made in his Paris pledge—but he was already busy undoing as many of those as he could. (Fortunately far fewer of these climate solutions than the media has implied are actually things Trump can reverse—we're already half-way to meeting our Paris pledge and Trump can't undo history). And since Paris is a coalition of the willing, not an enforcement based agreement, Trump was always free to walk away from our pledges and knew it.
So as a practical matter, withdrawing from Paris in 4 years will have no impact on U.S. climate emissions between now and 2025. Thursday's announcement doesn't give Trump a single additional tool to roll-back Obama's climate legacy.
So why do it? Why alienate virtually the entire global community, and abandon America's good standing as a diplomatic leader? Why let China and Germany step forward into our shoes? Why stiff more than 1,000 American businesses, including hundreds of the biggest, who begged Trump not to withdraw from Paris, including ExxonMobil and Chevron? Why make his climate denial such an embarrassingly big story?
It's hard to know exactly what motivates President Trump. My guess, though is that he withdrew from Paris precisely because he needed to show his isolationist, America- first followers that he would walk away from a passel of treaties, and that he rejected diplomacy as a tool of global leadership—in a way that would have fewer real world repercussions than keeping his campaign promise to terminate North American Free Trade Agreement.
Trump ignored the fact that the Paris accord was, for the U.S., an unbelievably favorable deal, in which everything we agreed to do was something most Americans wanted to do anyway: replace outmoded, expensive and dirty coal power with cheaper, cleaner renewables; stop wasting valuable natural gas by letting it leak or be flared; provide motorists with cars and trucks and waste less fuel and go further on a dollar's worth of gasoline; modernize our building stock to reduce utility bills and increase comfort; and replace climate destructive HFC refrigerants with modern, American developed safe alternatives.
His false and inflammatory remarks about the financial implications of the Paris agreement were clearly aimed at the minority of Americans who believe that global cooperation hurts the U.S., and displayed Trump's usual callousness about what most Americans hear or think about him—in this case, they are reading once again he is just not telling the truth. (For example, U.S. support for the Green Climate Fund under Paris is a tiny fraction of the $100 billion Trump cited. And nothing in the Paris agreement hampers the U.S.'s ability to build any particular energy project it chooses).
But Trump's entire campaign and Presidency have been premised on the notion that the passion of the minority which supports him will enable him to govern, even as the majority rejects his leadership—so this is nothing new.
What is new, however, is the intensity of the response. Many feared—and Steve Bannon hoped—that a U.S. withdrawal would create a domino collapse of global confidence in Paris. Instead it greatly solidified the commitment of Europe, China, Canada and India to filling the gap left by American withdrawal, while isolating and marginalizing the U.S. in other key diplomatic forums. India's Narendra Modi commented, "Paris or no Paris, our commitment to preserving the climate is for the sake of future generations."
Domestically, Trump has forced a large swathe of American business, which had welcomed the predictability and global consistency of the Paris agreement, into public opposition to his administration, a break companies had been very reluctant to risk. Elon Musk, who had controversially clung to his seat as one of Trump's economic advisors, walked out in protest. Jeff Immelt tweeted, "Industry must now lead and not depend on government." The Governors of California, Washington and New York began assembling a new coalition of states willing to challenge Trump and coordinate their climate leadership, expecting at least 10, and perhaps as many as 25 participants. Former New York Mayor Michael Bloomberg was pulling together a broad coalition of cities, states and the private sector to prepare an "American Pledge," a replacement set of strategies to meet America's Paris commitment to a 26 percent emissions cut, using state and local policy tools and corporate commitments, all aggregated and formally reported to the United Nations in place of the federal government reports previously envisaged. American society is replacing the Trump administration in climate diplomacy—something unprecedented but very powerful, and something which begins to marginalize the President in a disruptive and one expects unwelcome way.
This kind of a pathway to climate progress was laid out and anticipated by Mike Bloomberg and myself in our recent book, Climate of Hope. But we never imagined that it would be Donald Trump whose desire to remain center-stage for his base would accelerate and jump-start the process.
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By Ana Maldonado-Contreras
- Your gut is home to trillions of bacteria that are vital for keeping you healthy.
- Some of these microbes help to regulate the immune system.
- New research, which has not yet been peer-reviewed, shows the presence of certain bacteria in the gut may reveal which people are more vulnerable to a more severe case of COVID-19.
You may not know it, but you have an army of microbes living inside of you that are essential for fighting off threats, including the virus that causes COVID-19.
How Do Resident Bacteria Keep You Healthy?<p>Our immune defense is part of a complex biological response against harmful pathogens, such as viruses or bacteria. However, because our bodies are inhabited by trillions of mostly beneficial bacteria, virus and fungi, activation of our immune response is tightly regulated to distinguish between harmful and helpful microbes.</p><p>Our bacteria are spectacular companions diligently helping prime our immune system defenses to combat infections. A seminal study found that mice treated with antibiotics that eliminate bacteria in the gut exhibited an impaired immune response. These animals had low counts of virus-fighting white blood cells, weak antibody responses and poor production of a protein that is vital for <a href="https://doi.org/10.1073/pnas.1019378108" target="_blank">combating viral infection and modulating the immune response</a>.</p><p><a href="https://doi.org/10.1371/journal.pone.0184976" target="_blank" rel="noopener noreferrer">In another study</a>, mice were fed <em>Lactobacillus</em> bacteria, commonly used as probiotic in fermented food. These microbes reduced the severity of influenza infection. The <em>Lactobacillus</em>-treated mice did not lose weight and had only mild lung damage compared with untreated mice. Similarly, others have found that treatment of mice with <em>Lactobacillus</em> protects against different <a href="https://doi.org/10.1038/srep04638" target="_blank" rel="noopener noreferrer">subtypes of</a> <a href="https://doi.org/10.1038/s41598-017-17487-8" target="_blank" rel="noopener noreferrer">influenza</a> <a href="https://doi.org/10.1371/journal.ppat.1008072" target="_blank" rel="noopener noreferrer">virus</a> and human respiratory syncytial virus – the <a href="https://doi.org/10.1038/s41598-019-39602-7" target="_blank" rel="noopener noreferrer">major cause of viral bronchiolitis and pneumonia in children</a>.</p>
Chronic Disease and Microbes<p>Patients with chronic illnesses including Type 2 diabetes, obesity and cardiovascular disease exhibit a hyperactive immune system that fails to recognize a harmless stimulus and is linked to an altered gut microbiome.</p><p>In these chronic diseases, the gut microbiome lacks bacteria that activate <a href="https://doi.org/10.1126/science.1198469" target="_blank" rel="noopener noreferrer">immune cells</a> that block the response against harmless bacteria in our guts. Such alteration of the gut microbiome is also observed in <a href="https://doi.org/10.1073/pnas.1002601107" target="_blank" rel="noopener noreferrer">babies delivered by cesarean section</a>, individuals consuming a poor <a href="https://doi.org/10.1038/nature12820" target="_blank" rel="noopener noreferrer">diet</a> and the <a href="https://doi.org/10.1038/nature11053" target="_blank" rel="noopener noreferrer">elderly</a>.</p><p>In the U.S., 117 million individuals – about half the adult population – <a href="https://health.gov/our-work/food-nutrition/2015-2020-dietary-guidelines/guidelines/" target="_blank" rel="noopener noreferrer">suffer from Type 2 diabetes, obesity, cardiovascular disease or a combination of them</a>. That suggests that half of American adults carry a faulty microbiome army.</p><p>Research in my laboratory focuses on identifying gut bacteria that are critical for creating a balanced immune system, which fights life-threatening bacterial and viral infections, while tolerating the beneficial bacteria in and on us.</p><p>Given that diet affects the diversity of bacteria in the gut, <a href="https://www.umassmed.edu/nutrition/melody-trial-info/" target="_blank" rel="noopener noreferrer">my lab studies show how diet can be used</a> as a therapy for chronic diseases. Using different foods, people can shift their gut microbiome to one that boosts a healthy immune response.</p><p>A fraction of patients infected with SARS-CoV-2, the virus that causes COVID-19 disease, develop severe complications that require hospitalization in intensive care units. What do many of those patients have in common? <a href="https://www.cdc.gov/mmwr/volumes/69/wr/mm6912e2.htm" target="_blank" rel="noopener noreferrer">Old age</a> and chronic diet-related diseases like obesity, Type 2 diabetes and cardiovascular disease.</p><p><a href="http://doi.org/10.1016/j.jada.2008.12.019" target="_blank" rel="noopener noreferrer">Black and Latinx people are disproportionately affected by obesity, Type 2 diabetes and cardiovascular disease</a>, all of which are linked to poor nutrition. Thus, it is not a coincidence that <a href="https://www.cdc.gov/mmwr/volumes/69/wr/mm6933e1.htm" target="_blank" rel="noopener noreferrer">these groups have suffered more deaths from COVID-19</a> compared with whites. This is the case not only in the U.S. but also <a href="https://www.washingtonpost.com/world/europe/blacks-in-britain-are-four-times-as-likely-to-die-of-coronavirus-as-whites-data-show/2020/05/07/2dc76710-9067-11ea-9322-a29e75effc93_story.html" target="_blank" rel="noopener noreferrer">in Britain</a>.</p>
Discovering Microbes That Predict COVID-19 Severity<p>The COVID-19 pandemic has inspired me to shift my research and explore the role of the gut microbiome in the overly aggressive immune response against SARS-CoV-2 infection.</p><p>My colleagues and I have hypothesized that critically ill SARS-CoV-2 patients with conditions like obesity, Type 2 diabetes and cardiovascular disease exhibit an altered gut microbiome that aggravates <a href="https://theconversation.com/exercise-may-help-reduce-risk-of-deadly-covid-19-complication-ards-136922" target="_blank" rel="noopener noreferrer">acute respiratory distress syndrome</a>.</p><p>Acute respiratory distress syndrome, a life-threatening lung injury, in SARS-CoV-2 patients is thought to develop from a <a href="http://doi.org/10.1016/j.cytogfr.2020.05.003" target="_blank" rel="noopener noreferrer">fatal overreaction of the immune response</a> called a <a href="https://theconversation.com/blocking-the-deadly-cytokine-storm-is-a-vital-weapon-for-treating-covid-19-137690" target="_blank" rel="noopener noreferrer">cytokine storm</a> <a href="http://doi.org/10.1016/S2213-2600(20)30216-2" target="_blank" rel="noopener noreferrer">that causes an uncontrolled flood</a> <a href="http://doi.org/10.1016/S2213-2600(20)30216-2" target="_blank" rel="noopener noreferrer">of immune cells into the lungs</a>. In these patients, their own uncontrolled inflammatory immune response, rather than the virus itself, causes the <a href="http://doi.org/10.1007/s00134-020-05991-x" target="_blank" rel="noopener noreferrer">severe lung injury and multiorgan failures</a> that lead to death.</p><p>Several studies <a href="https://doi.org/10.1016/j.trsl.2020.08.004" target="_blank" rel="noopener noreferrer">described in one recent review</a> have identified an altered gut microbiome in patients with COVID-19. However, identification of specific bacteria within the microbiome that could predict COVID-19 severity is lacking.</p><p>To address this question, my colleagues and I recruited COVID-19 hospitalized patients with severe and moderate symptoms. We collected stool and saliva samples to determine whether bacteria within the gut and oral microbiome could predict COVID-19 severity. The identification of microbiome markers that can predict the clinical outcomes of COVID-19 disease is key to help prioritize patients needing urgent treatment.</p><p><a href="https://doi.org/10.1101/2021.01.05.20249061" target="_blank" rel="noopener noreferrer">We demonstrated</a>, in a paper which has not yet been peer reviewed, that the composition of the gut microbiome is the strongest predictor of COVID-19 severity compared to patient's clinical characteristics commonly used to do so. Specifically, we identified that the presence of a bacterium in the stool – called <em>Enterococcus faecalis</em>– was a robust predictor of COVID-19 severity. Not surprisingly, <em>Enterococcus faecalis</em> has been associated with <a href="https://doi.org/10.1053/j.gastro.2011.05.035" target="_blank" rel="noopener noreferrer">chronic</a> <a href="https://doi.org/10.1016/S0002-9440(10)61172-8" target="_blank" rel="noopener noreferrer">inflammation</a>.</p><p><em>Enterococcus faecalis</em> collected from feces can be grown outside of the body in clinical laboratories. Thus, an <em>E. faecalis</em> test might be a cost-effective, rapid and relatively easy way to identify patients who are likely to require more supportive care and therapeutic interventions to improve their chances of survival.</p><p>But it is not yet clear from our research what is the contribution of the altered microbiome in the immune response to SARS-CoV-2 infection. A recent study has shown that <a href="https://doi.org/10.1101/2020.12.11.416180" target="_blank" rel="noopener noreferrer">SARS-CoV-2 infection triggers an imbalance in immune cells</a> called <a href="https://doi.org/10.1111/imr.12170" target="_blank" rel="noopener noreferrer">T regulatory cells that are critical to immune balance</a>.</p><p>Bacteria from the gut microbiome are responsible for the <a href="https://doi.org/10.7554/eLife.30916.001" target="_blank" rel="noopener noreferrer">proper activation</a> <a href="https://doi.org/10.1126/science.1198469" target="_blank" rel="noopener noreferrer">of those T-regulatory</a> <a href="https://doi.org/10.1038/nri.2016.36" target="_blank" rel="noopener noreferrer">cells</a>. Thus, researchers like me need to take repeated patient stool, saliva and blood samples over a longer time frame to learn how the altered microbiome observed in COVID-19 patients can modulate COVID-19 disease severity, perhaps by altering the development of the T-regulatory cells.</p><p>As a Latina scientist investigating interactions between diet, microbiome and immunity, I must stress the importance of better policies to improve access to healthy foods, which lead to a healthier microbiome. It is also important to design culturally sensitive dietary interventions for Black and Latinx communities. While a good-quality diet might not prevent SARS-CoV-2 infection, it can treat the underlying conditions related to its severity.</p><p><em><a href="https://theconversation.com/profiles/ana-maldonado-contreras-1152969" target="_blank">Ana Maldonado-Contreras</a> is an assistant professor of Microbiology and Physiological Systems at the University of Massachusetts Medical School.</em></p><p><em>Disclosure statement: Ana Maldonado-Contreras receives funding from The Helmsley Charitable Trust and her work has been supported by the American Gastroenterological Association. She received The Charles A. King Trust Postdoctoral Research Fellowship. She is also member of the Diversity Committee of the American Gastroenterological Association.</em></p><p><em style="">Reposted with permission from <a href="https://theconversation.com/a-healthy-microbiome-builds-a-strong-immune-system-that-could-help-defeat-covid-19-145668" target="_blank" rel="noopener noreferrer" style="">The Conversation</a>. </em></p>
By Jeff Masters, Ph.D.
The New Climate War: the fight to take back our planet is the latest must-read book by leading climate change scientist and communicator Michael Mann of Penn State University.
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