
Natural Resources Defense Council
Have you ever wondered what chemicals might lurk in your couch? I did and so I decided to participate in a study, published yesterday, which analyzed what kinds of flame retardant chemicals are being used in upholstered furniture. I cut out a one inch square from one of the cushions of my still relatively new couch, wrapped it in foil and then bagged it into a Ziploc before sending it off. I wasn’t sure what would be found, crossed my fingers and hoped for the best.
Now I know—and the news isn’t good.
The foam in my couch contains the toxic flame retardant chemical, chlorinated Tris. And it isn’t just a small amount, because my couch is relatively big and has a lot of cushions. I have more than a pound, of this cancer-causing chemical in my living room. More than 35 years ago, chlorinated Tris was banned from kid’s pajamas because it was linked to cancer. Now it is in my couch, likely in my house dust, and in my body and in everyone in my family.
My couch wasn’t the only one. Out of 102 couches, the study found 85 percent contained toxic or untested flame retardants in the foam and all couches from California, except one purchased in 1989, contained measurable levels of at least one flame retardant chemical.
Chlorinated Tris, the chemical in my couch, was the most frequently found flame retardant chemical in the couches tested. Since the 1970s, chlorinated Tris has been recognized as a mutagen which damages DNA and can cause cancer. Chlorinated Tris is now listed under California’s Prop 65 as a carcinogen. As of October 2012, any products containing this chemical have to carry a warning label. In other words, if I was buying my couch today, it would likely carry a warning that looks like this:
Would you buy a couch with this label? I surely wouldn’t have if I knew then what I know now.
The second most commonly detected flame retardant chemical in this study was pentaBDE, which is recognized as a hormone disrupting chemical that interferes with development of the brain and nervous system and interferes with reproduction. Exposure to this chemical is associated with human health effects like delayed physical development, lower IQ, poorer attention, longer time to pregnancy and altered levels of sex hormones. PentaBDE was phased out of use in the U.S. in 2005, but because we hold onto our furniture for many years, many of couches with this chemical are still being used in American households. And PentaBDE is very persistent, so long after the exposure has ended, the chemical can remain in the body.
The third most frequently detected chemical was a proprietary mixture with the trade name of FireMaster 550. This mixture of chemicals appears to be the latest replacement flame retardant chemical in furniture with increasing use in furniture. FireMaster 550 has not been thoroughly tested but there are a number of concerns about it. One of the ingredients is a brominated form of the phthalate, DEHP, which is a well-recognized hormone disrupting chemical that has been shown to cause genital birth defects and cancer in laboratory animals. A recent study of FM 550 showed that small doses triggered obesity, anxiety and developmental problems in baby rats.
How did a chemical banned in pajamas get in my couch?
Unfortunately, it was only banned from pajamas and no other use. Under the current law, it is perfectly legal for this chemical to be in my couch. In the U.S., chemicals are presumed to be safe until found harmful, and the U.S. Environmental Protection Agency has little power to ban even notoriously deadly chemicals like asbestos. The reason toxic chemicals, like chlorinated Tris, have been allowed to remain on the market thirty years after they were recognized as carcinogens is also because of the failing of TSCA.
New legislation, the Safe Chemicals Act, would update and reform the Toxic Substances Control Act (TSCA) of 1976 and has been introduced by Senator Lautenberg of New Jersey. The proposed legislation being considered will give EPA more power to regulate the use of dangerous chemicals, including flame retardants.
Why are Californian’s couches the most polluted?
California has a unique flammability standard, TB 117, which applies to all upholstered furniture sold in the state. Couches sold in California, often contain a label that looks like this:
TB 117 is a performance standard which requires that the foam material withstand an open flame for 12 seconds. Although the standard doesn’t require the use of chemicals, the cheapest way to meet it has been to saturate furniture foam with toxic flame retardant chemicals. Millions of pounds of toxic chemicals are used for just this purpose, every year. And because couches aren’t just made for California, flame retardants were found in nearly all of the couches tested.
The trouble is—as well-intentioned as the standard may be—it has never been proven to be effective and as a recent hard hitting investigative series by the Chicago Tribune revealed, the flame retardant chemical industry worked with the tobacco industry to use deception, lots of money and lies to perpetuate the myth of the efficacy of these chemicals. Improved building codes requiring smoke detectors and water sprinklers, self-extinguishing cigarettes, and overall decreased rates of smoking have had a much bigger impact on the fewer number of fires and fire deaths observed over the past few decades.
The study found that more than 90 percent of couches purchased outside California during the last 7 years contained flame retardants, whether or not they carried a TB 117 label. Nearly two-thirds of couches that didn’t have the TB 117 label were still found to contain toxic or untested flame retardant chemicals. Demonstrating for the first time that we cannot rely on the presence or absence of a TB117 label to tell us if our couch is safe.
The good news is that California is taking steps to update the antiquated TB117 standard that is driving flame retardant use in furniture across the country. Despite past heavy lobbying by chemical producers to preserve TB117, Governor Jerry Brown has called for revisions to the standard that would improve fire safety without toxic flame retardants. If changes are successfully made, it may be possible to buy safe, non-toxic furniture by next summer.
We know that flame retardants don't stay put in our couches but migrate out and attach to our house dust which we either inhale or ingest. Another study published this week found more than forty flame retardant chemicals in California household dust, with the most frequently found being—you guessed it—chlorinated Tris.
So while we wait for the new furniture regulations to be put in place, we can reduce our exposure to these chemicals by reducing our dust exposure.
- Vacuum often (with an HEPA filter) and wet-mop to reduce build-up of dust in your home.
- Dust with a damp cloth or a microfiber cloth to avoid kicking up dust particles in the air as you work. For example, don’t use a feather duster as this only releases dust particles into the air.
- Wash hands frequently, (with plain soap and water!) as hand-to-mouth contact with dust is a major pathway for exposure.
And as for me, no more eating on the couch!
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A Healthy Microbiome Builds a Strong Immune System That Could Help Defeat COVID-19
By Ana Maldonado-Contreras
Takeaways
- Your gut is home to trillions of bacteria that are vital for keeping you healthy.
- Some of these microbes help to regulate the immune system.
- New research, which has not yet been peer-reviewed, shows the presence of certain bacteria in the gut may reveal which people are more vulnerable to a more severe case of COVID-19.
You may not know it, but you have an army of microbes living inside of you that are essential for fighting off threats, including the virus that causes COVID-19.
How Do Resident Bacteria Keep You Healthy?
<p>Our immune defense is part of a complex biological response against harmful pathogens, such as viruses or bacteria. However, because our bodies are inhabited by trillions of mostly beneficial bacteria, virus and fungi, activation of our immune response is tightly regulated to distinguish between harmful and helpful microbes.</p><p>Our bacteria are spectacular companions diligently helping prime our immune system defenses to combat infections. A seminal study found that mice treated with antibiotics that eliminate bacteria in the gut exhibited an impaired immune response. These animals had low counts of virus-fighting white blood cells, weak antibody responses and poor production of a protein that is vital for <a href="https://doi.org/10.1073/pnas.1019378108" target="_blank">combating viral infection and modulating the immune response</a>.</p><p><a href="https://doi.org/10.1371/journal.pone.0184976" target="_blank" rel="noopener noreferrer">In another study</a>, mice were fed <em>Lactobacillus</em> bacteria, commonly used as probiotic in fermented food. These microbes reduced the severity of influenza infection. The <em>Lactobacillus</em>-treated mice did not lose weight and had only mild lung damage compared with untreated mice. Similarly, others have found that treatment of mice with <em>Lactobacillus</em> protects against different <a href="https://doi.org/10.1038/srep04638" target="_blank" rel="noopener noreferrer">subtypes of</a> <a href="https://doi.org/10.1038/s41598-017-17487-8" target="_blank" rel="noopener noreferrer">influenza</a> <a href="https://doi.org/10.1371/journal.ppat.1008072" target="_blank" rel="noopener noreferrer">virus</a> and human respiratory syncytial virus – the <a href="https://doi.org/10.1038/s41598-019-39602-7" target="_blank" rel="noopener noreferrer">major cause of viral bronchiolitis and pneumonia in children</a>.</p>Chronic Disease and Microbes
<p>Patients with chronic illnesses including Type 2 diabetes, obesity and cardiovascular disease exhibit a hyperactive immune system that fails to recognize a harmless stimulus and is linked to an altered gut microbiome.</p><p>In these chronic diseases, the gut microbiome lacks bacteria that activate <a href="https://doi.org/10.1126/science.1198469" target="_blank" rel="noopener noreferrer">immune cells</a> that block the response against harmless bacteria in our guts. Such alteration of the gut microbiome is also observed in <a href="https://doi.org/10.1073/pnas.1002601107" target="_blank" rel="noopener noreferrer">babies delivered by cesarean section</a>, individuals consuming a poor <a href="https://doi.org/10.1038/nature12820" target="_blank" rel="noopener noreferrer">diet</a> and the <a href="https://doi.org/10.1038/nature11053" target="_blank" rel="noopener noreferrer">elderly</a>.</p><p>In the U.S., 117 million individuals – about half the adult population – <a href="https://health.gov/our-work/food-nutrition/2015-2020-dietary-guidelines/guidelines/" target="_blank" rel="noopener noreferrer">suffer from Type 2 diabetes, obesity, cardiovascular disease or a combination of them</a>. That suggests that half of American adults carry a faulty microbiome army.</p><p>Research in my laboratory focuses on identifying gut bacteria that are critical for creating a balanced immune system, which fights life-threatening bacterial and viral infections, while tolerating the beneficial bacteria in and on us.</p><p>Given that diet affects the diversity of bacteria in the gut, <a href="https://www.umassmed.edu/nutrition/melody-trial-info/" target="_blank" rel="noopener noreferrer">my lab studies show how diet can be used</a> as a therapy for chronic diseases. Using different foods, people can shift their gut microbiome to one that boosts a healthy immune response.</p><p>A fraction of patients infected with SARS-CoV-2, the virus that causes COVID-19 disease, develop severe complications that require hospitalization in intensive care units. What do many of those patients have in common? <a href="https://www.cdc.gov/mmwr/volumes/69/wr/mm6912e2.htm" target="_blank" rel="noopener noreferrer">Old age</a> and chronic diet-related diseases like obesity, Type 2 diabetes and cardiovascular disease.</p><p><a href="http://doi.org/10.1016/j.jada.2008.12.019" target="_blank" rel="noopener noreferrer">Black and Latinx people are disproportionately affected by obesity, Type 2 diabetes and cardiovascular disease</a>, all of which are linked to poor nutrition. Thus, it is not a coincidence that <a href="https://www.cdc.gov/mmwr/volumes/69/wr/mm6933e1.htm" target="_blank" rel="noopener noreferrer">these groups have suffered more deaths from COVID-19</a> compared with whites. This is the case not only in the U.S. but also <a href="https://www.washingtonpost.com/world/europe/blacks-in-britain-are-four-times-as-likely-to-die-of-coronavirus-as-whites-data-show/2020/05/07/2dc76710-9067-11ea-9322-a29e75effc93_story.html" target="_blank" rel="noopener noreferrer">in Britain</a>.</p>Discovering Microbes That Predict COVID-19 Severity
<p>The COVID-19 pandemic has inspired me to shift my research and explore the role of the gut microbiome in the overly aggressive immune response against SARS-CoV-2 infection.</p><p>My colleagues and I have hypothesized that critically ill SARS-CoV-2 patients with conditions like obesity, Type 2 diabetes and cardiovascular disease exhibit an altered gut microbiome that aggravates <a href="https://theconversation.com/exercise-may-help-reduce-risk-of-deadly-covid-19-complication-ards-136922" target="_blank" rel="noopener noreferrer">acute respiratory distress syndrome</a>.</p><p>Acute respiratory distress syndrome, a life-threatening lung injury, in SARS-CoV-2 patients is thought to develop from a <a href="http://doi.org/10.1016/j.cytogfr.2020.05.003" target="_blank" rel="noopener noreferrer">fatal overreaction of the immune response</a> called a <a href="https://theconversation.com/blocking-the-deadly-cytokine-storm-is-a-vital-weapon-for-treating-covid-19-137690" target="_blank" rel="noopener noreferrer">cytokine storm</a> <a href="http://doi.org/10.1016/S2213-2600(20)30216-2" target="_blank" rel="noopener noreferrer">that causes an uncontrolled flood</a> <a href="http://doi.org/10.1016/S2213-2600(20)30216-2" target="_blank" rel="noopener noreferrer">of immune cells into the lungs</a>. In these patients, their own uncontrolled inflammatory immune response, rather than the virus itself, causes the <a href="http://doi.org/10.1007/s00134-020-05991-x" target="_blank" rel="noopener noreferrer">severe lung injury and multiorgan failures</a> that lead to death.</p><p>Several studies <a href="https://doi.org/10.1016/j.trsl.2020.08.004" target="_blank" rel="noopener noreferrer">described in one recent review</a> have identified an altered gut microbiome in patients with COVID-19. However, identification of specific bacteria within the microbiome that could predict COVID-19 severity is lacking.</p><p>To address this question, my colleagues and I recruited COVID-19 hospitalized patients with severe and moderate symptoms. We collected stool and saliva samples to determine whether bacteria within the gut and oral microbiome could predict COVID-19 severity. The identification of microbiome markers that can predict the clinical outcomes of COVID-19 disease is key to help prioritize patients needing urgent treatment.</p><p><a href="https://doi.org/10.1101/2021.01.05.20249061" target="_blank" rel="noopener noreferrer">We demonstrated</a>, in a paper which has not yet been peer reviewed, that the composition of the gut microbiome is the strongest predictor of COVID-19 severity compared to patient's clinical characteristics commonly used to do so. Specifically, we identified that the presence of a bacterium in the stool – called <em>Enterococcus faecalis</em>– was a robust predictor of COVID-19 severity. Not surprisingly, <em>Enterococcus faecalis</em> has been associated with <a href="https://doi.org/10.1053/j.gastro.2011.05.035" target="_blank" rel="noopener noreferrer">chronic</a> <a href="https://doi.org/10.1016/S0002-9440(10)61172-8" target="_blank" rel="noopener noreferrer">inflammation</a>.</p><p><em>Enterococcus faecalis</em> collected from feces can be grown outside of the body in clinical laboratories. Thus, an <em>E. faecalis</em> test might be a cost-effective, rapid and relatively easy way to identify patients who are likely to require more supportive care and therapeutic interventions to improve their chances of survival.</p><p>But it is not yet clear from our research what is the contribution of the altered microbiome in the immune response to SARS-CoV-2 infection. A recent study has shown that <a href="https://doi.org/10.1101/2020.12.11.416180" target="_blank" rel="noopener noreferrer">SARS-CoV-2 infection triggers an imbalance in immune cells</a> called <a href="https://doi.org/10.1111/imr.12170" target="_blank" rel="noopener noreferrer">T regulatory cells that are critical to immune balance</a>.</p><p>Bacteria from the gut microbiome are responsible for the <a href="https://doi.org/10.7554/eLife.30916.001" target="_blank" rel="noopener noreferrer">proper activation</a> <a href="https://doi.org/10.1126/science.1198469" target="_blank" rel="noopener noreferrer">of those T-regulatory</a> <a href="https://doi.org/10.1038/nri.2016.36" target="_blank" rel="noopener noreferrer">cells</a>. Thus, researchers like me need to take repeated patient stool, saliva and blood samples over a longer time frame to learn how the altered microbiome observed in COVID-19 patients can modulate COVID-19 disease severity, perhaps by altering the development of the T-regulatory cells.</p><p>As a Latina scientist investigating interactions between diet, microbiome and immunity, I must stress the importance of better policies to improve access to healthy foods, which lead to a healthier microbiome. It is also important to design culturally sensitive dietary interventions for Black and Latinx communities. While a good-quality diet might not prevent SARS-CoV-2 infection, it can treat the underlying conditions related to its severity.</p><p><em><a href="https://theconversation.com/profiles/ana-maldonado-contreras-1152969" target="_blank">Ana Maldonado-Contreras</a> is an assistant professor of Microbiology and Physiological Systems at the University of Massachusetts Medical School.</em></p><p><em>Disclosure statement: Ana Maldonado-Contreras receives funding from The Helmsley Charitable Trust and her work has been supported by the American Gastroenterological Association. She received The Charles A. King Trust Postdoctoral Research Fellowship. She is also member of the Diversity Committee of the American Gastroenterological Association.</em></p><p><em style="">Reposted with permission from <a href="https://theconversation.com/a-healthy-microbiome-builds-a-strong-immune-system-that-could-help-defeat-covid-19-145668" target="_blank" rel="noopener noreferrer" style="">The Conversation</a>. </em></p>By Jeff Masters, Ph.D.
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