Michael Mann: Climate Change Worsened the Impact of Hurricane Harvey
Texas National Guardsmen assist residents affected by flooding caused by Hurricane Harvey onto a military vehicle in Houston, Aug. 27. Army National Guard / Lt. Zachary West
There are certain climate change-related factors that we can, with great confidence, say worsened the flooding.
Sea level rise attributable to climate change (some is due to coastal subsidence due to human disturbance e.g. oil drilling) is more than half a foot over the past few decades.
That means that the storm surge was a half foot higher than it would have been just decades ago, meaning far more flooding and destruction.
In addition to that, sea surface temperatures in the region have risen about 0.5C (close to 1F) over the past few decades, from roughly 30C (86F) to 30.5C (87F), which contributed to the very warm sea surface temperatures (30.5-31 C or 87-88F). There is a simple thermodynamic relationship known as the Clausius-Clapeyron equation that tells us there is a roughly 3 percent increase in average atmospheric moisture content for each 0.5C (~1F) of warming. Sea surface temperatures in the area where Harvey intensified were 0.5-1C warmer than current-day average temperatures, which translates to 1-1.5C warmer than the "average" temperatures a few decades ago. That means 3-5 percent more moisture in the atmosphere.
That large amount of moisture meant the potential for much greater rainfalls and greater flooding.
The combination of coastal flooding and heavy rainfall is responsible for the devastating flooding that Houston is experiencing.
Not only are the surface waters of the Gulf unusually warm right now, but there is a deep layer of warm water that Harvey was able to feed upon when it intensified at near record pace as it neared the coast. Human-caused warming is penetrating down into the ocean warming not just the surface but creating deeper layers of warm water in the Gulf and elsewhere.
So Harvey was almost certainly more intense than it would have been in the absence of human- caused warming, which means stronger winds, more wind damage and a larger storm surge (as an example of how this works, we have shown that climate change has led to a dramatic increase in storm surge risk in New York City, making devastating events like Superstorm Sandy more likely.
Finally, the more tenuous but potentially relevant climate factors: Part of what has made Harvey such a devastating storm is the way it has stalled right near the coast, continuing to pummel Houston and surrounding regions with a seemingly endless deluge which will likely top out at nearly 4 feet of rainfall over a several days-long period before it is done.
The stalling is due to very weak prevailing winds which are failing to steer the storm off to sea, allowing it to spin around and wobble back and forth like a top with no direction. This pattern, in turn, is associated with a greatly expanded subtropical high pressure system over much of the U.S. right now, with the jet stream pushed well to the north. This pattern of subtropical expansion is predicted in model simulations of human-caused climate change.
More tenuous, but possibly relevant still, is the fact that very persistent, nearly "stationary" summer weather patterns of this sort, where weather anomalies (both high pressure dry hot regions and low-pressure stormy/rainy regions) stay locked in place for many days at a time, appears to be favored by human-caused climate change. We recently published on this phenomenon:
In conclusion, while we cannot say climate change "caused" hurricane Harvey (that is an ill-posed question), we can say that it exacerbate several characteristics of the storm in a way that greatly increased the risk of damage and loss of life.
Climate change worsened the impact of Hurricane Harvey.
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An All-Rounder<p>Vitamin D levels in the body rise and fall according to sun exposure. If sufficient UV rays reach the skin, the body is able to produce the vitamin itself. However, the human body only derives an estimated 10 to 20 percent of its daily requirement from food.</p><p>The vitamin D that we synthesize from sunlight or food is not biologically active at first. Before the kidneys can produce the biologically active form of the vitamin, known as calcitriol, and release it into the blood, some metabolic processes must take place beforehand.</p><p>In addition, many organs have receptors to which the precursor of calcitriol binds. Further, this substance is also present in blood.</p><p>From this precursor, the organs then produce calcitriol themselves, which the body then uses for countless other processes in the body. This form of vitamin D thus regulates insulin secretion, inhibits tumor growth, and promotes the formation of red blood cells as well as the survival and activity of macrophages, which are important for the <a href="https://www.mdpi.com/2072-6643/5/7/2502/htm" target="_blank">immune system.</a></p>
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Association Versus Intervention Studies<p>Many studies on the vitamin are association or observational studies. "By definition, these studies cannot prove the causal relationship, but only point to mere correlations," said Fassnacht. The physician tries to illustrate this with an example:</p><p>"Imagine two groups of 80-year-olds. One group is spry, active and does sports. If you compare them with another group living in nursing homes, the difference in vitamin D levels will be dramatic. Life expectancy would also be extremely different."</p><p>But to try to explain the difference in fitness by vitamin D status alone is far too simplistic. "Vitamin D levels are a good measure of how sick someone is. But not more," says Fassnacht. </p><p>According to Fassnacht, none of the intervention studies carried out to date -- that specifically examined the effect of vitamin D on various diseases -- has been able to confirm the previous association and laboratory studies or the presumed positive effect of vitamin D.</p>
Further Research Is Needed<p>"If a coronavirus infection is suspected, it is therefore absolutely necessary to check the vitamin D status and quickly correct any possible deficit," said the recommendation of the paper published by the University of Hohenheim.</p><p>"Studies are underway to see whether vitamin D helps in COVID-19 infection, but I personally do not believe that this is really the case," says endocrinologist Fassnacht. Nevertheless, he says it is of course useful to carry out these studies.<br></p><p>"I don't want to rule out that there are actually subgroups of people who benefit from an additional vitamin D dose," he says. After all, this has been proven to be the case with a severe deficit.</p><p>In view of the study situation, Fassnacht does not think much of preventive, nationwide vitamin D substitutes. "My belief that the vitamin helps somewhere is very low. But, of course, I can be wrong."</p>
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