Today, nearly 65 billion animals worldwide, including cows, chickens and pigs, are crammed into CAFOs.
A growing number of organic consumers, natural health advocates and climate hawks are taking a more comprehensive look at the fundamental causes of global warming. And its led them to this sobering conclusion: our modern energy-, chemical- and genetically modified organism (GMO)-intensive industrial food and farming systems are the major cause of man-made global warming.
How did they reach this conclusion? First, by taking a more inclusive look at the scientific data on greenhouse gas (GHG) emissions—not just carbon dioxide (CO2), but also methane and nitrous oxide. Next, by doing a full accounting of the fossil fuel consumption and emissions of the entire industrial food and farming cycle, including inputs, equipment, production, processing, distribution, heating, cooling and waste. And finally, by factoring in the indirect impacts of contemporary agriculture, which include deforestation and wetlands destruction.
When you add it all up, the picture is clear—contemporary agriculture is burning up our planet. And factory farms or, in industry lingo, Confined Animal Feeding Operations (CAFOs), play a key role in this impending disaster.
The science behind global warming is complex. Without question, coal plants, tar sands and natural gas fracking have contributed heavily to GHG pollution, the major cause of global warming. We must unite to shut down these industries. Similarly, consumer overconsumption of fossil fuels represents another big piece of the climate-crisis equation. We absolutely must rethink, retrofit and/or redesign our gas-guzzling cars and our energy-inefficient buildings, if we want to reduce fossil fuel use by 90 percent over the next few decades.
But we also must address the environmental impact of factory farming.
Today, nearly 65 billion animals worldwide, including cows, chickens and pigs, are crammed into CAFOs. These animals are literally imprisoned and tortured in unhealthy, unsanitary and unconscionably cruel conditions. Sickness is the norm for animals who are confined rather than pastured, and who eat GMO corn and soybeans, rather than grass and forage as nature intended. To prevent the inevitable spread of disease from stress, overcrowding and lack of vitamin D, animals are fed a steady diet of antibiotics. Those antibiotics pose a direct threat to the environment when they run off into our lakes, rivers, aquifers and drinking water.
CAFOs contribute directly to global warming by releasing vast amounts of greenhouse gases into the atmosphere—more than the entire global transportation industry. The air at some factory farm test sites in the U.S. is dirtier than in America’s most polluted cities, according to the Environmental Integrity Project. According to a 2006 report by the Food and Agriculture Organization of the United Nations (FAO), animal agriculture is responsible for 18 percent of all human-induced greenhouse gas emissions, including 37 percent of methane emissions and 65 percent of nitrous oxide emissions. The methane releases from billions of imprisoned animals on factory farms are 70 times more damaging per ton to the earth’s atmosphere than CO2.
Indirectly, factory farms contribute to climate disruption by their impact on deforestation and draining of wetlands, and because of the nitrous oxide emissions from huge amounts of pesticides used to grow the genetically engineered corn and soy fed to animals raised in CAFOs. Nitrous oxide pollution is even worse than methane—200 times more damaging per ton than CO2. And just as animal waste leaches antibiotics and hormones into ground and water, pesticides and fertilizers also eventually find their way into our waterways, further damaging the environment.
Factory farms aren’t just a disaster for the environment. They’re also ruining our health. A growing chorus of scientists and public health advocates warn that the intensive and reckless use of antibiotics and growth hormones leads to factory-farmed food that contains antibiotic-resistant pathogens, drug residues such as hormones and growth promoters, and “bad fats.” Yet despite these health and environmental hazards, the vast majority of consumers don’t realize that nearly 95 percent of the meat, dairy and eggs sold in the U.S. come from CAFOs. Nor do most people realize that CAFOs represent a corporate-controlled system characterized by large-scale, centralized, low profit-margin production, processing and distribution systems.
There’s an alternative: a socially responsible, small-scale system created by independent producers and processors focused on local and regional markets. This alternative produces high-quality food, and supports farmers who produce healthy, meat, eggs and dairy products using humane methods.
And it’s far easier on the environment.
Consumers can boycott food products from factory farms and choose the more environmentally-friendly alternatives. But first, we have to regain the right to know what’s in our food. And that means mandatory labeling, not only of genetically engineered foods, but of the 95 percent of non-organic, non-grass-fed meat, dairy and eggs that are produced on the hellish factory farms that today dominate U.S. food production.
In 2013, a new alliance of organic and natural health consumers, animal welfare advocates, anti-GMO and climate-change activists will tackle the next big food labeling battle: meat, eggs and dairy products from animals raised on factory farms, or CAFOs. This campaign will start with a massive program to educate consumers about the negative impacts of factory farming on the environment, on human health and on animal welfare, and then move forward to organize and mobilize millions of consumers to demand labels on beef, pork, poultry and dairy products derived from these unhealthy and unsustainable so-called “farming” practices.
Opponents and skeptics will ask, “What about feeding the world?” Contrary to popular arguments, factory farming is not a cheap, efficient solution to world hunger. Feeding huge numbers of confined animals actually uses more food, in the form of grains that could feed humans, than it produces. For every 100 food calories of edible crops fed to livestock, we get back just 30 calories in the form of meat and dairy. That’s a 70-percent loss.
With the earth’s population predicted to reach nine billion by mid-century, the planet can no longer afford this reckless, unhealthy and environmentally disastrous farming system. We believe that once people know the whole truth about CAFOs they will want to make healthier, more sustainable food choices. And to do that, we’ll have to fight for the consumer’s right to know not only what is in our food, but where our food comes from.
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By Jacob L. Steenwyk and Antonis Rokas
From the mythical minotaur to the mule, creatures created from merging two or more distinct organisms – hybrids – have played defining roles in human history and culture. However, not all hybrids are as fantastic as the minotaur or as dependable as the mule; in fact, some of them cause human diseases.
When Looking Through a Microscope Isn’t Close Enough.<p>For the last few years, <a href="http://www.rokaslab.org/" target="_blank">our team at Vanderbilt University</a>, <a href="https://www.researchgate.net/lab/Gustavo-Goldman-Lab" target="_blank">Gustavo Goldman's team at São Paulo University in Brazil</a> and many other collaborators around the world have been collecting samples of fungi from patients infected with different species of <em>Aspergillus</em> molds. One of the species we are particularly interested in is <a href="https://doi.org/10.1006/rwgn.2001.0082" target="_blank"><em>Aspergillus nidulans</em>, a relatively common and generally harmless fungus</a>. Clinical laboratories typically identify the species of <em>Aspergillus</em> causing the infection by examining cultures of the fungi under the microscope. The problem with this approach is that very closely related species of <em>Aspergillus</em> tend to look very similar in their broad morphology or physical appearance when viewing them through a microscope.</p><p>Interested in examining the varying abilities of different <em>A. nidulans</em> strains to cause disease, we decided to analyze their total genetic content, or genomes. What we saw came as a total surprise. We had not collected <em>A. nidulans</em> but <em>Aspergillus latus</em>, a close relative of <em>A. nidulans</em> and, as we were to soon find out, <a href="https://doi.org/10.1016/j.cub.2020.04.071" target="_blank">a hybrid species that evolved through the fusion of the genomes</a> of two other <em>Aspergillus</em> species: <em>Aspergillus spinulosporus</em> and an unknown close relative of <em>Aspergillus quadrilineatus</em>. Thus, we realized not only that these patients harbored infections from an entirely different species than we thought they were, but also that this species was the first ever <em>Aspergillus</em> hybrid known to cause human infections.</p>
Several Different Fungal Hybrids Cause Human Disease.<p>Hybrid fungi that can cause infections in humans are well known to occur in several different lineages of single-celled fungi known as yeasts. Notable examples include multiple different species of <a href="https://doi.org/10.1002/yea.3242" target="_blank">yeast hybrids</a> that cause the human diseases <a href="https://rarediseases.info.nih.gov/diseases/6218/cryptococcosis" target="_blank">cryptococcosis</a> and <a href="https://www.cdc.gov/fungal/diseases/candidiasis/index.html" target="_blank">candidiasis</a>. Although pathogenic yeast hybrids are well known, our discovery that the <em>A. latus</em> pathogen is a hybrid is a first for molds that cause disease in humans.</p>
(Left) Candida yeasts live on parts of the human body. Imbalance of microbes on the body can allow these yeasts, some of which are hybrids, to grow and cause infection. (Right) Cryptococcus yeasts, including ones that are hybrids, can cause life-threatening infections in primarily immunocompromised people. Centers for Disease Control and Prevention<p><a href="https://doi.org/10.1371/journal.ppat.1008315" target="_blank">Why certain <em>Aspergillus</em> species are so deadly</a> while others are harmless remains unknown. This may in part be because <a href="https://doi.org/10.1016/j.fbr.2007.02.007" target="_blank">combinations of traits, rather than individual traits</a>, underlie organisms' ability to cause disease. So why then are hybrids frequently associated with human disease? Hybrids inherit genetic material from both parents, which may result in new combinations of traits. This may make them more similar to one parent in some of their characteristics, reflect both parents in others or may differ from both in the rest. It is precisely this mix and match of traits that hybrids have inherited from their parental species that <a href="https://www.nytimes.com/2010/09/14/science/14creatures.html" target="_blank">facilitates their evolutionary success</a>, including their ability to cause disease.</p>
The Evolutionary Origin of an Aspergillus Hybrid.<p>Multiple evolutionary paths can lead to the emergence of hybrids. One path is through mating, just as the horse and donkey mate to create a mule. Another path is through the merging or fusion of genetic material from cells of different species.</p><p>It is this second path that appears to have been taken by our fungus. <em>A. latus</em> appears to have two of almost everything compared to its parental species: twice the genome size, twice the total number of genes and so on. But unlike other hybrids, which are often sterile like the mule, we found that <em>A. latus</em> is capable of reproducing both asexually and sexually.</p><p>But how distinct were the parents of <em>A. latus</em>? By comparing the parts contributed by each parent in the <em>A. latus</em> genome, we estimate that its parents are approximately 93% genetically similar, which is about as related as we humans are with lemurs. In other words, <em>A. latus</em>, an agent of infectious disease, is the fungal equivalent of a human-lemur hybrid.</p>
How A. Latus Differs From its Parents.<p>Elucidating the identity of closely related fungal pathogens and how they differ from each other in infection-relevant characteristics is a key step toward reducing the burden of fungal disease. For example, we found that <em>A. latus</em> was three times more resistant than <em>A. nidulans</em>, the species it was originally identified as using microscopy-based methods, to one of the most common antifungal drugs, <a href="https://www.drugbank.ca/drugs/DB00520" target="_blank">caspofungin</a>. This result provides a clear example of the potential importance of accurate identification of the <em>Aspergillus</em> pathogen causing an infection.</p><p>We also examined how <em>A. latus</em> and <em>A. nidulans</em> interact with cells from our immune system. We found that immune cells were less efficient at combating <em>A. latus</em> compared to <em>A. nidulans</em>, suggesting the hybrid fungus may be trickier for our immune systems to identify and destroy.</p><p>In the midst of the COVID-19 pandemic, our quest to understand <em>Aspergillus</em> pathogens is becoming more urgent. Growing evidence suggests that <a href="https://doi.org/10.1111/myc.13096" target="_blank">a fraction of COVID-19 patients are also infected with <em>Aspergillus</em>.</a> More worrying is that these <a href="https://doi.org/10.3201/eid2607.201603" target="_blank">secondary <em>Aspergillus</em> infections</a> can worsen the clinical outcomes for those infected with the novel coronavirus. That being said, we stress that little is known about <em>Aspergillus</em> infections in COVID-19 patients due to a lack of systematic testing, and none of the infections identified so far appear to have been caused by hybrids.</p><p>So, when it comes to hybrids, some are fantastic (the minotaur), some are helpful (the mule) and some are dangerous (<em>Aspergillus latus</em>). Understanding more about the biology of <em>Aspergillus latus</em> may help in our understanding of how microbial pathogens arise and how to best prevent and combat their infections.</p>
This Saturday, June 6, marks National Trails Day, an annual celebration of the remarkable recreational, scenic and hiking trails that crisscross parks nationwide. The event, which started in 1993, honors the National Trail System and calls for volunteers to help with trail maintenance in parks across the country.
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The Navajo Nation covers the corners of three different states. Google Maps
Growing Contribution<img lazy-loadable="true" src="https://assets.rebelmouse.io/eyJhbGciOiJIUzI1NiIsInR5cCI6IkpXVCJ9.eyJpbWFnZSI6Imh0dHBzOi8vYXNzZXRzLnJibC5tcy8yMzM3NDY5Ny9vcmlnaW4ucG5nIiwiZXhwaXJlc19hdCI6MTY0NjM4MTgyM30.IuQTKQs1stvYYKD6vaVTrqAyoBsUG0BhDvlhxsyKwPA/img.png?width=980" id="02a05" class="rm-shortcode" data-rm-shortcode-id="2841f82b1785df5d5ed7bf64d3bb882b" data-rm-shortcode-name="rebelmouse-image" />
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DAN medical experts explained the difference between normal lungs, on the left, and "very serious lungs caused by COVID-19," on the right. Matias Nochetto / Divers Alert Network (DAN)
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