Climate Science Explained in These 10 Charts
By Kelly Levin
Thousands of people are expected to attend the People's Climate Movement march in Washington, DC and sister cities around the world this coming weekend. They are marching because actions taken to date by governments and others are not commensurate with the scale of climate impacts—both those already borne and those projected in the years to come.
It's a good moment to reflect on the facts. What do we know about global climate change and what impacts can we expect in the future? The following graphics speak volumes.
1. What is Climate Change?
Climate change is a long-term change in Earth's weather patterns or average climate, including temperature and precipitation. While the climate has changed in the past, we are now seeing it change at an unprecedented rate. As a result of the build-up of heat-trapping greenhouse gases in the atmosphere—due to our burning of fossil fuels, cutting down trees and other activities—global average temperature is now changing at a faster rate than at least over the past 1,000 years.
2. What's Causing Climate Change?
When models only include natural drivers of climate change, such as natural variability and volcanic eruptions, they cannot reproduce the recent increase in temperature. Only when models include the increase in greenhouse gas emissions due to human activities can they replicate the observed changes.
U.S. Enviromental Protection Agency, adapted from Huber and Knutti, 2012
3. How Have Global Emissions Changed?
Emissions have been climbing since the Industrial Revolution, but the rate of annual emissions increase during the first 10 years of this century was almost double the rate between 1970 and 2000.
Global Carbon Project
Emissions from fossil fuels and industry have seen a staggering increase in recent years—63 percent since 1990.
4. Who Are the Biggest Emitters?
From 1850 to 2011, the five major emitters—the U.S., European Union, China, Russian Federation and Japan— together contributed two-thirds of the world's CO2 emissions.
Now, China has emerged as the top emitter and China, the EU and the U.S. are the world's top three emitters. Together they emit more than half of total global greenhouse gases. In contrast, the 100 smallest-emitting countries collectively add up to only 3.5 percent of global emissions. Almost three-quarters of global emissions come from only 10 countries.
5. How Much Should We Limit Global Warming?
The Paris agreement on climate change sets a target for countries to collectively limit global temperature rise to 2 degrees C (3.6 degrees F), with a goal of sticking to 1.5 degrees C (2.7 degrees F) in order to prevent some of the worst effects of climate change. The amount of carbon emissions we can emit while still having a likely chance of limiting warming to 2 degrees is known as the "carbon budget." As of 2011, the world had already blown through nearly two-thirds of the carbon budget and is on track to exceed it by 2033 if emissions continue unabated.
6. Where is the Temperature Headed?
In the absence of countries' recent emissions-reduction commitments, known as intended nationally determined contributions or INDCs, we would see 4-5 degrees C of warming. Even if these INDCs are fully implemented, the average global temperature is still on track to increase 2.7-3.7 degrees C by 2100, according to a range of studies. That's far short of the global goal to limit warming to 1.5- 2 degrees C.
7. What Have Been Some of the Impacts of Climate Change to Date?
The impacts of climate change are already occurring and occurring everywhere. For example, climate change has already led to: more negative than positive impacts to crops, such as wheat and maize; coral bleaching and species range shifts; more frequent heat waves; coastal flooding; increased tree die-off in various regions; and a significant loss of ice mass in places like Greenland and Antarctica.
For example, as a result of ice melting on land, such as from glaciers and ice sheets, as well as thermal expansion of the ocean, we have seen sea level rise 3.4 millimeters per year from 1993-2015, which puts coastal communities at risk of flooding and infrastructure damage.
8. What Impacts Do We Expect in the Future?
The impacts we see in the future will be determined by our emissions pathway and resultant level of temperature increase. The warmer it gets, the greater the impacts—and the lower our ability to adapt.
9. Are There Signs of Progress?
Recently, we've seen signs of "decoupling." According to the International Energy Agency, energy-related carbon dioxide emissions stayed flat for three years in a row even as the global economy grew. This flattening of emissions was due to the growth of renewable power generation, fuel switching from coal to natural gas and energy efficiency gains, among other changes.
This decoupling can also be seen at the country level in 21 nations from 2000-2014. Whether these are indicative of long-term shifts remains to be seen. We will need to see a deep decline if we are to limit dangerous climate change and even with existing emissions-reduction commitments, global emissions are not expected to decline until at least after 2030.
20. Are We Investing in Solutions?
Global investments in renewable energy have been growing in recent years to an all-time high of $285.9 billion in 2015, a 5 percent rise compared to the previous year. In 2015, renewable energy (excluding large hydro) made up the bulk (54.6 percent) of new installed generating capacity for the first time.
REN21 Renewables 2016 Global Status Report
That being said, we need to shift away from fossil fuels much more quickly if we are to have a fighting chance of limiting warming to 1.5-2 degrees C.
Marching for Action
Let's hope that as people take to the streets, it will wake leaders up to the scale of the climate change challenge and the task ahead. Avoiding the most dangerous of climate change impacts—which necessitates phasing out emissions in the second half of the century—will require sustained action well beyond this weekend's activities.
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By Jacob L. Steenwyk and Antonis Rokas
From the mythical minotaur to the mule, creatures created from merging two or more distinct organisms – hybrids – have played defining roles in human history and culture. However, not all hybrids are as fantastic as the minotaur or as dependable as the mule; in fact, some of them cause human diseases.
When Looking Through a Microscope Isn’t Close Enough.<p>For the last few years, <a href="http://www.rokaslab.org/" target="_blank">our team at Vanderbilt University</a>, <a href="https://www.researchgate.net/lab/Gustavo-Goldman-Lab" target="_blank">Gustavo Goldman's team at São Paulo University in Brazil</a> and many other collaborators around the world have been collecting samples of fungi from patients infected with different species of <em>Aspergillus</em> molds. One of the species we are particularly interested in is <a href="https://doi.org/10.1006/rwgn.2001.0082" target="_blank"><em>Aspergillus nidulans</em>, a relatively common and generally harmless fungus</a>. Clinical laboratories typically identify the species of <em>Aspergillus</em> causing the infection by examining cultures of the fungi under the microscope. The problem with this approach is that very closely related species of <em>Aspergillus</em> tend to look very similar in their broad morphology or physical appearance when viewing them through a microscope.</p><p>Interested in examining the varying abilities of different <em>A. nidulans</em> strains to cause disease, we decided to analyze their total genetic content, or genomes. What we saw came as a total surprise. We had not collected <em>A. nidulans</em> but <em>Aspergillus latus</em>, a close relative of <em>A. nidulans</em> and, as we were to soon find out, <a href="https://doi.org/10.1016/j.cub.2020.04.071" target="_blank">a hybrid species that evolved through the fusion of the genomes</a> of two other <em>Aspergillus</em> species: <em>Aspergillus spinulosporus</em> and an unknown close relative of <em>Aspergillus quadrilineatus</em>. Thus, we realized not only that these patients harbored infections from an entirely different species than we thought they were, but also that this species was the first ever <em>Aspergillus</em> hybrid known to cause human infections.</p>
Several Different Fungal Hybrids Cause Human Disease.<p>Hybrid fungi that can cause infections in humans are well known to occur in several different lineages of single-celled fungi known as yeasts. Notable examples include multiple different species of <a href="https://doi.org/10.1002/yea.3242" target="_blank">yeast hybrids</a> that cause the human diseases <a href="https://rarediseases.info.nih.gov/diseases/6218/cryptococcosis" target="_blank">cryptococcosis</a> and <a href="https://www.cdc.gov/fungal/diseases/candidiasis/index.html" target="_blank">candidiasis</a>. Although pathogenic yeast hybrids are well known, our discovery that the <em>A. latus</em> pathogen is a hybrid is a first for molds that cause disease in humans.</p>
(Left) Candida yeasts live on parts of the human body. Imbalance of microbes on the body can allow these yeasts, some of which are hybrids, to grow and cause infection. (Right) Cryptococcus yeasts, including ones that are hybrids, can cause life-threatening infections in primarily immunocompromised people. Centers for Disease Control and Prevention<p><a href="https://doi.org/10.1371/journal.ppat.1008315" target="_blank">Why certain <em>Aspergillus</em> species are so deadly</a> while others are harmless remains unknown. This may in part be because <a href="https://doi.org/10.1016/j.fbr.2007.02.007" target="_blank">combinations of traits, rather than individual traits</a>, underlie organisms' ability to cause disease. So why then are hybrids frequently associated with human disease? Hybrids inherit genetic material from both parents, which may result in new combinations of traits. This may make them more similar to one parent in some of their characteristics, reflect both parents in others or may differ from both in the rest. It is precisely this mix and match of traits that hybrids have inherited from their parental species that <a href="https://www.nytimes.com/2010/09/14/science/14creatures.html" target="_blank">facilitates their evolutionary success</a>, including their ability to cause disease.</p>
The Evolutionary Origin of an Aspergillus Hybrid.<p>Multiple evolutionary paths can lead to the emergence of hybrids. One path is through mating, just as the horse and donkey mate to create a mule. Another path is through the merging or fusion of genetic material from cells of different species.</p><p>It is this second path that appears to have been taken by our fungus. <em>A. latus</em> appears to have two of almost everything compared to its parental species: twice the genome size, twice the total number of genes and so on. But unlike other hybrids, which are often sterile like the mule, we found that <em>A. latus</em> is capable of reproducing both asexually and sexually.</p><p>But how distinct were the parents of <em>A. latus</em>? By comparing the parts contributed by each parent in the <em>A. latus</em> genome, we estimate that its parents are approximately 93% genetically similar, which is about as related as we humans are with lemurs. In other words, <em>A. latus</em>, an agent of infectious disease, is the fungal equivalent of a human-lemur hybrid.</p>
How A. Latus Differs From its Parents.<p>Elucidating the identity of closely related fungal pathogens and how they differ from each other in infection-relevant characteristics is a key step toward reducing the burden of fungal disease. For example, we found that <em>A. latus</em> was three times more resistant than <em>A. nidulans</em>, the species it was originally identified as using microscopy-based methods, to one of the most common antifungal drugs, <a href="https://www.drugbank.ca/drugs/DB00520" target="_blank">caspofungin</a>. This result provides a clear example of the potential importance of accurate identification of the <em>Aspergillus</em> pathogen causing an infection.</p><p>We also examined how <em>A. latus</em> and <em>A. nidulans</em> interact with cells from our immune system. We found that immune cells were less efficient at combating <em>A. latus</em> compared to <em>A. nidulans</em>, suggesting the hybrid fungus may be trickier for our immune systems to identify and destroy.</p><p>In the midst of the COVID-19 pandemic, our quest to understand <em>Aspergillus</em> pathogens is becoming more urgent. Growing evidence suggests that <a href="https://doi.org/10.1111/myc.13096" target="_blank">a fraction of COVID-19 patients are also infected with <em>Aspergillus</em>.</a> More worrying is that these <a href="https://doi.org/10.3201/eid2607.201603" target="_blank">secondary <em>Aspergillus</em> infections</a> can worsen the clinical outcomes for those infected with the novel coronavirus. That being said, we stress that little is known about <em>Aspergillus</em> infections in COVID-19 patients due to a lack of systematic testing, and none of the infections identified so far appear to have been caused by hybrids.</p><p>So, when it comes to hybrids, some are fantastic (the minotaur), some are helpful (the mule) and some are dangerous (<em>Aspergillus latus</em>). Understanding more about the biology of <em>Aspergillus latus</em> may help in our understanding of how microbial pathogens arise and how to best prevent and combat their infections.</p>
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By John Letzing
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The Navajo Nation covers the corners of three different states. Google Maps
Growing Contribution<img lazy-loadable="true" src="https://assets.rebelmouse.io/eyJhbGciOiJIUzI1NiIsInR5cCI6IkpXVCJ9.eyJpbWFnZSI6Imh0dHBzOi8vYXNzZXRzLnJibC5tcy8yMzM3NDY5Ny9vcmlnaW4ucG5nIiwiZXhwaXJlc19hdCI6MTY0NjM4MTgyM30.IuQTKQs1stvYYKD6vaVTrqAyoBsUG0BhDvlhxsyKwPA/img.png?width=980" id="02a05" class="rm-shortcode" data-rm-shortcode-id="2841f82b1785df5d5ed7bf64d3bb882b" data-rm-shortcode-name="rebelmouse-image" />
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DAN medical experts explained the difference between normal lungs, on the left, and "very serious lungs caused by COVID-19," on the right. Matias Nochetto / Divers Alert Network (DAN)
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